Busty sleeps and is still fine? Thanks for your lucky genes

A boy, some people have all the genetic luck. In a recent study, scientists describe a newly discovered boom that allows humans to sleep naturally much less hours per night without any negative health effects.

The researchers in China conducted the study, Published In the facts of the National Academy of Science. A boom was found in a 70 -year -old healthy woman who lived her entire life barely sleeping. Researchers say the results can one day help us understand how to treat sleep disturbances that affect the rest of humanity.

Most people need about seven to nine hours of sleep per night for optimal health. But there is some of the so -called natural sleep, who are able to rest between four and six hours every night without experimenting with the symptoms of sleep deprivation (it should not be confused with a third of the people who Sleep is less than seven hours per night and suffers for that). Research has found that these individuals tend to carry unique genetic mutations.

To date, scientists have found mutations in Four different genes (DEC2, Npsr1, GRM1, and AdRB1) linked to natural short sleep. But researchers behind this new study have now found another: Kinaz 3 or SIK3 gene, the gene, which was called the name of the protein that it makes. SIK3 is protein quinaz, which is a type of enzyme. It is believed to play a role in metabolism, but previous research (Mostly in miceHe also suggested that it helps to affect the duration of sleep.

The team analyzed the patterns of sleep and DNA of the volunteers. Although it usually reported only three hours of sleep per night, ACTIGRAPY (movement at night is usually captured via WristWatch) found that it was already sleeping 6.3 hours in the average average. The team’s genetic exploration also identified a specific mutation in the SIK3 gene – formed N783Y – which seems to explain its natural short sleep.

To confirm their discovery, they classified the mice with the same boom, and found that they slept less than regular mice. The mutation seems to prevent the SIK3 ability to transfer certain particles to other proteins as usual, especially the important proteins of neurons, which are the ties that form between neurons.

The authors wrote: “These results emphasize the function saved by SIK3 as a critical prisoner in regulating human sleep.”

maybe Only 1 % Among the world’s population is natural asleep. But the lessons that we learn to reveal their unique genetic gift can help scientists find new goals to treat sleep disorders. The study researchers have already found some evidence that other protein quinators similar to SIK3 also play a role in influencing our sleep.

“These results enhance our understanding of the genetic foundations of sleep, highlighting the broader effects of Kenaz’s activity in regulating sleep across species, and providing more support for possible treatment strategies to enhance sleep efficiency,” they wrote.

In any case, hoping that scientists will also discover the genetic cause that my cat must wake me up at 6 am rain or sunlight, although knowing that breakfast is hours.