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A virus may have long played that it is not harmful to a role in Parkinson’s disease, A case affects more than a million Americans.
Western Shamali medicine scientists have discovered HPGV in the brains of the backbone of people with Parkinson’s, but not in those who have no disease. The results challenge contracts of assumptions about the virus.
“HPGV is a common infection that was not known in the past that was not often known to affect the brain often,” said Dr. Igor Corralic, head of neurological diseases in the northwest.
“We were surprised to find him in the brains of Parkinson’s patients in such a high frequency and not in controls.”
The researchers examined the brain tissue after death from 10 Parkinson’s patients and 14 people who died for other reasons. (Istock)
The results were published in JCI Insight.
The researchers examined the brain tissue after death from 10 Parkinson’s patients and 14 people who died for other reasons. The virus appeared in five Barkinson’s ten brains and was none of the 14 controls.
It also appeared in the samples of the spine, indicating that the virus can be active in Nervous system. Patients with HPGV showed more progressive changes in the brain associated with Parkinson, including protein accumulation and changing brain chemistry.
The team did not stop at the brain tissue. Using blood samples from more than 1000 participants in a project led by Michael J. Fox, the researchers saw the same changes in the immune system associated with the virus.
The researchers also examined the blood samples of 1,000 people with Parkinson’s, trying to track the virus. (Istock)
The most surprising thing: patients with Parkinson’s mutation associated with the LRRK2 gene differently from HPGV from those who have no mutation.
“This indicates that it may be an environmental factor that interacts with the body in ways that we have not realized before,” said Coralnick.
“This may affect how Parkinson’s developed, especially in people who have some Genetic backgrounds“
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Parkinson’s disease is the second most common brain disorder after Alzheimer’s disease, according to the National Institute for Neurological Disorders and Stroke. While a small percentage of cases inherited, most patients do not have a family history, and the reason remains unknown.
“It is too early to say that the virus causes the disease,” says Dr. Joel Salinas, a behavioral neurologist. (Istock)
If HPGV is really playing a role, this may help to clarify the reason for the development of some people in Parkinson while others do not do it. It can also open the door to new treatments targeting viruses or immune system.
“The study has discovered the effects of HPGV often in the brains of people with Parkinson’s disease more than controls. This raises the possibility of a link between viral exposure and Parkinson’s paralysis, but it is too early to say Virus “It causes disease,” Dr. Joel Salinas, a behavioral neurologist and associate professor at the New York University Medical College, told Fox News Digital.
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Salinas, who did not participate in the study, also said that there will be much larger and long -term studies to determine whether this bond is of real clinical importance.
“At the present time, people should know that this is early research and not a cause of anxiety – there is no overwhelming evidence yet that this virus causes Parkinson’s, similar to how working in herpes viruses in Alzheimer’s disease suggested possible but to be fully established.”
The Northweestern team is planning to expand their studies to find out the number of times that HPGV is located in people with Parkinson. (Istock)
According to the Parkinson Foundation, approximately 90,000 Americans are diagnosed every year. This number is expected to reach 1.2 million by 2030.
NorthWestern team is planning to expand their studies to find out the number of times HPGV is located in people with Parkinson’s controls against health controls, and whether other viruses may be involved.
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“As for the virus, which he believed was harmless, these results indicate that it may have important effects in the context of Parkinson’s disease,” said Coralnick.
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“We also aim to understand how viruses and genes interact; visions that can reveal how Parkinson’s begins and can help direct future treatments.”
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